Transmembrane protein 150b
Appearance
TMEM150B | |||||||||||||||||||||||||||||||||||||||||||||||||||
---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|---|
Identifiers | |||||||||||||||||||||||||||||||||||||||||||||||||||
Aliases | TMEM150B, TMEM224, transmembrane protein 150B, TTN2, DRAM3 | ||||||||||||||||||||||||||||||||||||||||||||||||||
External IDs | OMIM: 617291; MGI: 2679718; HomoloGene: 19603; GeneCards: TMEM150B; OMA:TMEM150B - orthologs | ||||||||||||||||||||||||||||||||||||||||||||||||||
| |||||||||||||||||||||||||||||||||||||||||||||||||||
| |||||||||||||||||||||||||||||||||||||||||||||||||||
| |||||||||||||||||||||||||||||||||||||||||||||||||||
| |||||||||||||||||||||||||||||||||||||||||||||||||||
Wikidata | |||||||||||||||||||||||||||||||||||||||||||||||||||
|
Transmembrane protein 150B is a protein that in humans is encoded by the TMEM150B gene.[5]
Function
The protein belongs to the DRAM (damage-regulated autophagy modulator) family of membrane-spanning proteins. Alternate splicing results in multiple transcript variants. [provided by RefSeq, Aug 2013].
References
- ^ a b c GRCh38: Ensembl release 89: ENSG00000180061 – Ensembl, May 2017
- ^ a b c GRCm38: Ensembl release 89: ENSMUSG00000046456 – Ensembl, May 2017
- ^ "Human PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
- ^ "Mouse PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
- ^ "Entrez Gene: Transmembrane protein 150B". Retrieved 2018-05-07.
Further reading
- Mrschtik M, O'Prey J, Lao LY, Long JS, Beaumatin F, Strachan D, O'Prey M, Skommer J, Ryan KM (October 2015). "DRAM-3 modulates autophagy and promotes cell survival in the absence of glucose". Cell Death Differ. 22 (10): 1714–26. doi:10.1038/cdd.2015.26. PMC 4563785. PMID 25929859.
This article incorporates text from the United States National Library of Medicine, which is in the public domain.